What can result from excessive levels of aldosterone in Conn's syndrome?

In Conn's syndrome, which is characterized by excessive production of aldosterone from the adrenal glands, one of the primary physiological impacts is the retention of sodium and water, leading to an expansion of the blood volume. Elevated aldosterone levels promote sodium reabsorption in the kidneys while increasing potassium excretion. As a consequence of this sodium and fluid retention, blood pressure rises, resulting in hypertension.

Hypertension associated with Conn's syndrome can lead to various cardiovascular issues over time if not managed properly. Therefore, the correct answer reflects the primary consequence of excessive aldosterone production in this syndrome. On the other hand, the other options do not align with the effects seen in Conn's syndrome; hypotension would typically indicate low blood volume or low sodium levels, bradycardia relates more to heart rate issues that are not directly caused by aldosterone levels, and hyperkalemia is unlikely as increased aldosterone typically leads to lower potassium levels due to increased renal excretion.